Search results for "Inflammasome activation"

showing 4 items of 4 documents

Targeting the Endoplasmic Reticulum Unfolded Protein Response to Counteract the Oxidative Stress-Induced Endothelial Dysfunction

2017

In endothelial cells, the tight control of the redox environment is essential for the maintenance of vascular homeostasis. The imbalance between ROS production and antioxidant response can induce endothelial dysfunction, the initial event of many cardiovascular diseases. Recent studies have revealed that the endoplasmic reticulum could be a new player in the promotion of the pro- or antioxidative pathways and that in such a modulation, the unfolded protein response (UPR) pathways play an essential role. The UPR consists of a set of conserved signalling pathways evolved to restore the proteostasis during protein misfolding within the endoplasmic reticulum. Although the first outcome of the U…

0301 basic medicineAgingProgrammed cell deathendocrine systemOxidative phosphorylationReview Articlemedicine.disease_causeEndoplasmic ReticulumBiochemistryINITIATION-FACTOR 2-ALPHA03 medical and health sciencesProgrammed cell-deathSELECTIVE-INHIBITIONProgrammed cell-death;TXNIP/NLRP3 INFLAMMASOME ACTIVATION; MITOCHONDRIAL ELECTRON-TRANSPORT; SPONTANEOUSLY HYPERTENSIVE-RATS; INITIATION-FACTOR 2-ALPHA; CORONARY-ARTERY FUNCTION; ER STRESS; SELECTIVE-INHIBITION; MESSENGER-RNA; TRANSMEMBRANE PROTEINmedicineHumansEndothelial dysfunctionlcsh:QH573-671TXNIP/NLRP3 INFLAMMASOME ACTIVATIONSPONTANEOUSLY HYPERTENSIVE-RATSEndothelial Cellbusiness.industrylcsh:CytologyEndoplasmic reticulumfungiEndothelial CellsOxidative StreCell BiologyGeneral MedicineAdaptive responseMITOCHONDRIAL ELECTRON-TRANSPORTER STRESSmedicine.diseaseCell biologyOxidative Stress030104 developmental biologyProteostasisTRANSMEMBRANE PROTEINUnfolded protein responseUnfolded Protein ResponsebusinessMESSENGER-RNAOxidative stressCORONARY-ARTERY FUNCTIONHumanOxidative Medicine and Cellular Longevity
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Phytochemical inhibitors of the NLRP3 inflammasome for the treatment of inflammatory diseases

2021

The NLRP3 inflammasome holds a crucial role in innate immune responses. Pathogen- and danger-associated molecular patterns may initiate inflammasome activation and following inflammatory cytokine release. The inflammasome formation and its-associated activity are involved in various pathological conditions such as cardiovascular, central nervous system, metabolic, renal, inflammatory and autoimmune diseases. Although the mechanism behind NLRP3-mediated disorders have not been entirely illuminated, many phytochemicals and medicinal plants have been described to prevent inflammatory disorders. In the present review, we mainly introduced phytochemicals inhibiting NLRP3 inflammasome in addition…

0301 basic medicineInflammasomesmedicine.medical_treatmentPhytochemicalsAnti-Inflammatory AgentsInflammation03 medical and health sciences0302 clinical medicineNLR Family Pyrin Domain-Containing 3 ProteinmedicineAnimalsHumansInflammationPharmacologyInnate immune systemintegumentary systembusiness.industryMechanism (biology)Inflammasome030104 developmental biologyCytokinePhytochemical030220 oncology & carcinogenesisImmunologyNLRP3 inflammasome activationInflammation Mediatorsmedicine.symptomSignal transductionbusinessSignal Transductionmedicine.drugPharmacological Research
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Melatonin protects rats from radiotherapy-induced small intestine toxicity

2017

Radiotherapy-induced gut toxicity is among the most prevalent dose-limiting toxicities following radiotherapy. Prevention of radiation enteropathy requires protection of the small intestine. However, despite the prevalence and burden of this pathology, there are currently no effective treatments for radiotherapy-induced gut toxicity, and this pathology remains unclear. The present study aimed to investigate the changes induced in the rat small intestine after external irradiation of the tongue, and to explore the potential radio-protective effects of melatonin gel. Male Wistar rats were subjected to irradiation of their tongues with an X-Ray YXLON Y.Tu 320-D03 irradiator, receiving a dose o…

Male0301 basic medicineCancer TreatmentDrug Evaluation Preclinicallcsh:MedicineExpressionApoptosisToxicologyPathology and Laboratory Medicinemedicine.disease_causeBiochemistryOxidative Phosphorylation0302 clinical medicineIntestinal mucosaGastrointestinal tractIntestine SmallMedicine and Health SciencesRadiation-injuryIntestinal Mucosalcsh:ScienceEnergy-Producing OrganellesMelatoninCancerMultidisciplinaryNF-kappa BInflammasomeLipid-peroxidationGlutathioneMitochondriaRadiation therapyRadiation Injuries Experimentalmedicine.anatomical_structureOncology030220 oncology & carcinogenesisCancer treatmentToxicityInflammasome activationSmall IntestineExperimental pathologyAnatomyCellular Structures and OrganellesResearch Articlemedicine.drugClinical OncologyMucositismedicine.medical_specialtyRadiation TherapyRadiation-Protective AgentsBioenergeticsBiologyRadiation enteropathyMelatonin03 medical and health sciencesTongueInternal medicineSepsisNLR Family Pyrin Domain-Containing 3 ProteinmedicineAnimalsRats WistarMouthToxicitylcsh:RBiology and Life SciencesCell BiologySmall intestinemedicine.diseaseHormonesSmall intestinePathobiologyGastrointestinal TractOxidative Stress030104 developmental biologyEndocrinologylcsh:QClinical MedicineDigestive SystemGelsOxidative stress
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Clostridium difficile Induced Inflammasome Activation and Coagulation Derangements

2022

C. difficile enterocolitis (CDAC) is the most common hospital infection, burdened by an increased incidence of coagulation-related complications such as deep vein thrombosis (DVT) and disseminated intravascular coagulation (DIC) as well as a significant sepsis-related mortality. In this review, we analyzed the available data concerning the correlation between coagulation complications related to C. difficile infection (CDI) and inflammasome activation, in particular the pyrin-dependent one. The little but solid available preclinical and clinical evidence shows that inflammasome activation increases the risk of venous thromboembolism (VTE). As proof of this, it has been observed that in vitr…

sepsisMicrobiology (medical)enterocolitiinduced inflammasome activationcoagulation derangementVirologyClostridium difficileMicrobiologyMicroorganisms
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